DNA Research, Genetics and Cell Biology by Tsisana Shartava

By Tsisana Shartava

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Actually, six PAH specifically present in the Prestige oil were classified as 32 Beatriz Pérez-Cadahía, Josefina Méndez, Eduardo Pásaro et al. possible or probable human carcinogens by the IARC, and were included among the 16 PAH designated by the USEPA as primary contaminants: benzo[a]anthracene, benzo[b]fluoranthene, benzo[k]fluoranthene, benzo[a]pyrene, dibenz[ah]anthracene and indeno[1,2,3-cd]pyrene (Figure 2). Nowadays PAH are considered as procarcinogens and tumour inductors. , 1993). , 1998).

Targeted point mutations of p53 lead to dominant-negative inhibition ofwild-type p53 function. Proc. Natl. Acad. Sci. 2002; 99: 2948-53. [109] Schmitt CA, Fridman JS, Yang M, Baranov E, Hoffman RM, Lowe SW. Dissecting p53 tumor suppressor functions in vivo. Cancer Cell 2002; 1: 289-98. [110] Brooks CI, Gu W. Ubiquitination, phosphorylation and acetylation. The molecular basis for p53 regulation. Curr. Opin. Cell Biol. 2003; 15: 164-71. [111] Pfeifer GP, p53 mutational spectra and the role of methylated CpG sequences Mutat.

Deregulated c-Myc prematurely recruits both Type I and II CD95/Fas apoptotic pathways associated with terminal myeloid differentiation. Oncogene 2002; 21(10): 1600-10. [103] Chen YQ, Zhou YQ, Wang MH. Activation of the RON receptor tyrosine kinase protects murine macrophages from apoptotic death induced by bacterial lipopolysaccharide. J. Leukoc Biol. 2002 ;71(2): 359-66. [104] Scian MJ, Stagliano KE, Anderson MA, Hassan S, Bowman M, Miles MF, Deb SP. Tumor-derived p53 mutants induce NF-kappaB2 gene expression.

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